Autophagy protein ATG5 interacts transiently with the hepatitis C virus RNA polymerase (NS5B) early during infection
- 15 September 2010
- journal article
- research article
- Published by Elsevier BV in Virology
- Vol. 405 (1), 1-7
- https://doi.org/10.1016/j.virol.2010.05.032
Abstract
No abstract availableKeywords
Funding Information
- NSERC (312225-05)
- Armand-Frappier Foundation (Canada)
This publication has 34 references indexed in Scilit:
- Novel HCV replication mouse model using human hepatocellular carcinoma xenograftsAntiviral Research, 2009
- Formation and function of hepatitis C virus replication complexes require residues in the carboxy-terminal domain of NS4B proteinVirology, 2009
- The autophagy machinery is required to initiate hepatitis C virus replicationProceedings of the National Academy of Sciences of the United States of America, 2009
- Autophagosome Supports Coxsackievirus B3 Replication in Host CellsJournal of Virology, 2008
- Hepatitis C Virus Genotype 1a Growth and Induction of AutophagyJournal of Virology, 2008
- Structure of Atg5·Atg16, a Complex Essential for AutophagyPublished by Elsevier BV ,2007
- Molecular Mechanism of a Thumb Domain Hepatitis C Virus Nonnucleoside RNA-Dependent RNA Polymerase InhibitorAntimicrobial Agents and Chemotherapy, 2006
- Polypyrimidine-tract-binding Protein is a Component of the HCV RNA Replication Complex and Necessary for RNA SynthesisJournal of Biomedical Science, 2006
- Production of infectious hepatitis C virus in tissue culture from a cloned viral genomeNature Medicine, 2005
- Dissection of Autophagosome Formation Using Apg5-Deficient Mouse Embryonic Stem CellsThe Journal of cell biology, 2001