Opa1-Mediated Cristae Opening Is Bax/Bak and BH3 Dependent, Required for Apoptosis, and Independent of Bak Oligomerization
- 22 August 2008
- journal article
- Published by Elsevier BV in Molecular Cell
- Vol. 31 (4), 557-569
- https://doi.org/10.1016/j.molcel.2008.07.010
Abstract
No abstract availableFunding Information
- National Institutes of Health (GM50284, GM62289, 7028-05, DK54441, ES010337, RR04050)
- National Institute of Environmental Health Sciences (GM50284, GM62289, 7028-05, DK54441, ES010337, RR04050)
- Leukemia and Lymphoma Society (GM50284, GM62289, 7028-05, DK54441, ES010337, RR04050)
This publication has 32 references indexed in Scilit:
- Regulation of the mitochondrial dynamin-like protein Opa1 by proteolytic cleavageThe Journal of cell biology, 2007
- GAPDH and Autophagy Preserve Survival after Apoptotic Cytochrome c Release in the Absence of Caspase ActivationCell, 2007
- Mitochondrial Rhomboid PARL Regulates Cytochrome c Release during Apoptosis via OPA1-Dependent Cristae RemodelingCell, 2006
- OPA1 Controls Apoptotic Cristae Remodeling Independently from Mitochondrial FusionCell, 2006
- Mcl-1 Interacts with Truncated Bid and Inhibits Its Induction of Cytochrome c Release and Its Role in Receptor-mediated ApoptosisOnline Journal of Public Health Informatics, 2006
- Release of OPA1 during Apoptosis Participates in the Rapid and Complete Release of Cytochrome c and Subsequent Mitochondrial FragmentationOnline Journal of Public Health Informatics, 2005
- Oligomeric Bax Is a Component of the Putative CytochromecRelease Channel MAC, Mitochondrial Apoptosis-induced ChannelMolecular Biology of the Cell, 2005
- Loss of the Intermembrane Space Protein Mgm1/OPA1 Induces Swelling and Localized Constrictions along the Lengths of MitochondriaOnline Journal of Public Health Informatics, 2004
- The coordinate release of cytochrome c during apoptosis is rapid, complete and kinetically invariantNature, 2000
- Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarizationThe EMBO Journal, 1998