II. Correlations between secondary structure stability and mutation frequency during somatic hypermutation
- 31 August 2008
- journal article
- Published by Elsevier BV in Molecular Immunology
- Vol. 45 (13), 3600-3608
- https://doi.org/10.1016/j.molimm.2008.05.012
Abstract
No abstract availableKeywords
This publication has 12 references indexed in Scilit:
- Detection of chromatin-associated single-stranded DNA in regions targeted for somatic hypermutationThe Journal of Experimental Medicine, 2007
- Mechanisms of genotoxin-induced transcription and hypermutation in p53Cancer Cell International, 2006
- Intricate targeting of immunoglobulin somatic hypermutation maximizes the efficiency of affinity maturationThe Journal of Experimental Medicine, 2005
- Increased transcription rates correlate with increased reversion rates in leuB and argH Escherichia coli auxotrophsMicrobiology, 2004
- Mechanisms by which transcription can regulate somatic hypermutationGenes & Immunity, 2004
- Predicting mutation frequencies in stem-loop structures of derepressed genes: implications for evolutionMolecular Microbiology, 2003
- Hypermutable bases in the p53 cancer gene are at vulnerable positions in DNA secondary structures.2002
- The intrinsic hypermutability of antibody heavy and light chain genes decays exponentiallyThe EMBO Journal, 2001
- Spontaneous mutation in the Escherichia coli lacI gene.Genetics, 1991
- Mutation Drift and Repertoire Shift in the Maturation of the Immune ResponseImmunological Reviews, 1987