The growth compromised HSV-2 mutant ΔRR prevents kainic acid-induced apoptosis and loss of function in organotypic hippocampal cultures
- 13 November 2006
- journal article
- Published by Elsevier BV in Brain Research
- Vol. 1119 (1), 26-39
- https://doi.org/10.1016/j.brainres.2006.08.078
Abstract
No abstract availableThis publication has 70 references indexed in Scilit:
- Brain Response to Injury and Neurodegeneration: Endogenous Neuroprotective SignalingAnnals of the New York Academy of Sciences, 2005
- Expression levels of Rab2, a G protein, and Bag-1, a Bcl-2 binding protein are controlled by withdrawal of nicotine from cultured pheochromocytoma PC12 cellsJournal of Neural Transmission, 2005
- Chronic activation of ERK and neurodegenerative diseasesBioEssays, 2003
- The Herpes Simplex Virus Type 2 R1 Protein Kinase (ICP10 PK) Functions as a Dominant Regulator of Apoptosis in Hippocampal Neurons Involving Activation of the ERK Survival Pathway and Upregulation of the Antiapoptotic Protein Bag-1Journal of Virology, 2003
- Estrogen regulation of c-fos gene expression through phosphatidylinositol-3-kinase-dependent activation of serum response factor in MCF-7 breast cancer cellsBiochemical and Biophysical Research Communications, 2002
- HSV-1 Vector-Delivered FGF2 to the Retina Is Neuroprotective but Does Not Preserve Functional ResponsesMolecular Therapy, 2001
- Coupling of cAMP/PKA and MAPK Signaling in Neuronal Cells Is Dependent on Developmental StageExperimental Neurology, 2001
- Downregulation of Bim, a Proapoptotic Relative of Bcl-2, Is a Pivotal Step in Cytokine-Initiated Survival Signaling in Murine Hematopoietic ProgenitorsMolecular and Cellular Biology, 2001
- ATP and SH3 Binding Sites in the Protein Kinase of the Large Subunit of Herpes Simplex Virus Type 2 of Ribonucleotide Reductase (ICP10)Published by Elsevier BV ,1996
- Immediate early and functional AP-1 cis-response elements are involved in the transcriptional regulation of the large subunit of herpes simplex virus type 2 ribonucleotide reductase (ICP10)Virus Research, 1992