Curcumin eliminates oxidized LDL roles in activating hepatic stellate cells by suppressing gene expression of lectin-like oxidized LDL receptor-1
Open Access
- 1 November 2009
- journal article
- research article
- Published by Elsevier BV in Laboratory Investigation
- Vol. 89 (11), 1275-1290
- https://doi.org/10.1038/labinvest.2009.93
Abstract
No abstract availableThis publication has 66 references indexed in Scilit:
- Natural derivatives of curcumin attenuate the Wnt/β-catenin pathway through down-regulation of the transcriptional coactivator p300Biochemical and Biophysical Research Communications, 2008
- Mechanisms of Hepatic FibrogenesisGastroenterology, 2008
- Association Between Circulating Oxidized Low-Density Lipoprotein and Incidence of the Metabolic SyndromeJama-Journal Of The American Medical Association, 2008
- Activation of peroxisome proliferator-activated receptor-γ by curcumin blocks the signaling pathways for PDGF and EGF in hepatic stellate cellsLaboratory Investigation, 2008
- Canonical Wnt signaling maintains the quiescent stage of hepatic stellate cellsBiochemical and Biophysical Research Communications, 2008
- Curcumin: potential for hepatic fibrosis therapy?British Journal of Pharmacology, 2008
- De novo synthesis of glutathione is a prerequisite for curcumin to inhibit hepatic stellate cell (HSC) activationFree Radical Biology & Medicine, 2007
- Functional Interaction between Peroxisome Proliferator-Activated Receptor γ and β-CateninMolecular and Cellular Biology, 2006
- PPARγ regulates adipocyte cholesterol metabolism via oxidized LDL receptor 1JCI Insight, 2005
- PPARγ Ligands Inhibit TNF-α-Induced LOX-1 Expression in Cultured Endothelial CellsBiochemical and Biophysical Research Communications, 2001