Overrepresentation of the proarrhythmic, sudden death predisposing sodium channel polymorphism S1103Y in a population-based cohort of African-American sudden infant death syndrome
- 31 May 2008
- journal article
- Published by Elsevier BV in Heart Rhythm
- Vol. 5 (5), 712-715
- https://doi.org/10.1016/j.hrthm.2008.02.012
Abstract
No abstract availableThis publication has 23 references indexed in Scilit:
- Molecular and Functional Characterization of Novel Glycerol-3-Phosphate Dehydrogenase 1–Like Gene ( GPD1-L ) Mutations in Sudden Infant Death SyndromeCirculation, 2007
- A mechanism for sudden infant death syndrome (SIDS): Stress-induced leak via ryanodine receptorsHeart Rhythm, 2007
- Novel mechanism for sudden infant death syndrome: Persistent late sodium current secondary to mutations in caveolin-3Heart Rhythm, 2007
- Cardiac Sodium Channel Dysfunction in Sudden Infant Death SyndromeCirculation, 2007
- Role of SCN5A Y1102 Polymorphism in Sudden Cardiac Death in BlacksCirculation, 2005
- Near-miss SIDS due to Brugada syndromeArchives of Disease in Childhood, 2005
- Ethnic Differences in Cardiac Potassium Channel Variants: Implications for Genetic Susceptibility to Sudden Cardiac Death and Genetic Testing for Congenital Long QT SyndromeMayo Clinic Proceedings, 2003
- Molecular diagnosis in a child with sudden infant death syndromeThe Lancet, 2001
- Genomic Organization of the HumanSCN5AGene Encoding the Cardiac Sodium ChannelGenomics, 1996
- The contribution of changes in the prevalence of prone sleeping position to the decline in sudden infant death syndrome in TasmaniaJAMA, 1995