A model for experimental induction of acute temporomandibular joint inflammation in rats: Effects of substance P(SP) on neuropeptide-like immunoreactivity
- 6 September 1996
- journal article
- Published by Elsevier BV in Life Sciences
- Vol. 59 (15), 1193-1201
- https://doi.org/10.1016/0024-3205(96)00309-8
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- Relation between intra-articular temperature of the arthritic temporomandibular joint and presence of calcitonin gene-related peptide in the joint fluid: A clinical studyActa Odontologica Scandinavica, 1993
- Substance P Actions on Sensory NeuronsAnnals of the New York Academy of Sciences, 1991
- Substance P Afferents Regulate ACTH-Corticosterone ReleaseAnnals of the New York Academy of Sciences, 1991
- Concentration of Substance P, Neurokinin A, Calcitonin Gene-related Peptide, Neuropeptide Y and Vasoactive Intestinal Polypeptide in Synovial Fluid from Knee Joints in Patients Suffering from Rheumatoid ArthritisScandinavian Journal of Rheumatology, 1991
- Inhibition of mitogen-stimulated T lymphocyte proliferation by calcitonin gene-related peptideBiochemical and Biophysical Research Communications, 1988
- Sample handling techniques when analyzing regulatory peptidesLife Sciences, 1987
- Coexistence of peptides with classical neurotransmittersCellular and Molecular Life Sciences, 1987
- Vasoconstrictor effects in vivo and plasma disappearance rate of neuropeptide Y in manLife Sciences, 1987
- Calcitonin gene-related peptide is a potent vasodilatorNature, 1985
- Identification of catabolin, a protein from synovium which induces degradation of cartilage in organ cultureBiochemical and Biophysical Research Communications, 1980