The SH2 Domain-containing Inositol 5′-Phosphatase (SHIP) Recruits the p85 Subunit of Phosphoinositide 3-Kinase during FcγRIIb1-mediated Inhibition of B Cell Receptor Signaling
Open Access
- 1 March 1999
- journal article
- Published by Elsevier BV
- Vol. 274 (11), 7489-7494
- https://doi.org/10.1074/jbc.274.11.7489
Abstract
No abstract availableThis publication has 31 references indexed in Scilit:
- Phosphatidylinositol-3,4,5-trisphosphate (PtdIns-3,4,5-P3)/Tec kinase-dependent calcium signaling pathway: a target for SHIP-mediated inhibitory signalsThe EMBO Journal, 1998
- Direct Regulation of the Akt Proto-Oncogene Product by Phosphatidylinositol-3,4-bisphosphateScience, 1997
- Role of the inositol phosphatase SHIP in negative regulation of the immune system by the receptor FeγRIIBNature, 1996
- Multiple forms of an inositol polyphosphate 5-phosphatase form signaling complexes with Shc and Grb2Current Biology, 1996
- Life, death, and the pursuit of apoptosis.Genes & Development, 1996
- Bad, a heterodimeric partner for Bcl-xL and Bcl-2, displaces bax and promotes cell deathCell, 1995
- CD19 of B Cells as a Surrogate Kinase Insert Region to Bind Phosphatidylinositol 3-KinaseScience, 1993
- SH2 domains recognize specific phosphopeptide sequencesCell, 1993
- PDGF-dependent tyrosine phosphorylation stimulates production of novel polyphosphoinositides in intact cellsCell, 1989
- Crosslinking of surface immunoglobulin and Fc receptors on B lymphocytes inhibits stimulation of inositol phospholipid breakdown via the antigen receptors.The Journal of Experimental Medicine, 1985