Inhibition of hepatitis C virus core protein expression in immortalized human hepatocytes induces cytochrome c-independent increase in Apaf-1 and caspase-9 activation for cell death
- 18 April 2005
- journal article
- Published by Elsevier BV in Virology
- Vol. 336 (2), 198-207
- https://doi.org/10.1016/j.virol.2005.03.016
Abstract
No abstract availableKeywords
This publication has 42 references indexed in Scilit:
- Apaf-1 Is a Mediator of E2F-1-induced ApoptosisOnline Journal of Public Health Informatics, 2002
- The Bcl2 family: regulators of the cellular life-or-death switchNature Reviews Cancer, 2002
- Hepatitis C Virus Core Protein Is Necessary for the Maintenance of Immortalized Human HepatocytesVirology, 2002
- APAF1 is a key transcriptional target for p53 in the regulation of neuronal cell deathThe Journal of cell biology, 2001
- Hepatitis C Virus Core Protein Modulates the Interferon-Induced Transacting Factors of Jak/Stat Signaling Pathway But Does Not Affect the Activation of Downstream IRF-1 or 561 GeneVirology, 2001
- Hepatitis C virus core protein promotes cell proliferation through the upregulation of cyclin E expression levelsLiver International, 2001
- Caspase Activation Involves the Formation of the Aposome, a Large (∼700 kDa) Caspase-activating ComplexOnline Journal of Public Health Informatics, 1999
- p14ARF links the tumour suppressors RB and p53Nature, 1998
- IAPs block apoptotic events induced by caspase-8 and cytochrome c by direct inhibition of distinct caspasesThe EMBO Journal, 1998
- Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarizationThe EMBO Journal, 1998