Netrin-1 induces proliferation of Schwann cells through Unc5b receptor
- 3 November 2007
- journal article
- Published by Elsevier BV in Biochemical and Biophysical Research Communications
- Vol. 362 (4), 1057-1062
- https://doi.org/10.1016/j.bbrc.2007.08.143
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- Netrin-1 and its receptors in tumorigenesisNature Reviews Cancer, 2004
- Netrin-1 controls colorectal tumorigenesis by regulating apoptosisNature, 2004
- Semaphorin 3B (SEMA3B) induces apoptosis in lung and breast cancer, whereas VEGF 165 antagonizes this effectProceedings of the National Academy of Sciences of the United States of America, 2004
- Molecular control of neuronal migrationBioEssays, 2002
- Netrin-1 acts as a survival factor via its receptors UNC5H and DCCThe EMBO Journal, 2001
- Semaphorins as Mediators of Neuronal ApoptosisJournal of Neurochemistry, 1999
- RETRACTED: A Ligand-Gated Association between Cytoplasmic Domains of UNC5 and DCC Family Receptors Converts Netrin-Induced Growth Cone Attraction to RepulsionCell, 1999
- Vertebrate homologues of C. elegans UNC-5 are candidate netrin receptorsNature, 1997
- Deleted in Colorectal Cancer (DCC) Encodes a Netrin ReceptorCell, 1996
- Netrins are diffusible chemotropic factors for commissural axons in the embryonic spinal cordCell, 1994