Genetic Risk Can Be Decreased: Quitting Smoking Decreases and Delays Lung Cancer for Smokers With High and Low CHRNA5 Risk Genotypes — A Meta-Analysis
Open Access
- 10 August 2016
- journal article
- research article
- Published by Elsevier BV in EBioMedicine
- Vol. 11, 219-226
- https://doi.org/10.1016/j.ebiom.2016.08.012
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Method for Evaluating Multiple Mediators: Mediating Effects of Smoking and COPD on the Association between the CHRNA5-A3 Variant and Lung Cancer RiskPLOS ONE, 2012
- Interplay of Genetic Risk Factors (CHRNA5-CHRNA3-CHRNB4) and Cessation Treatments in Smoking Cessation SuccessAmerican Journal of Psychiatry, 2012
- CHRNA3 rs1051730 Genotype and Short-Term Smoking CessationNicotine & Tobacco Research, 2011
- Individual and Cumulative Effects of GWAS Susceptibility Loci in Lung Cancer: Associations after Sub-Phenotyping for COPDPLOS ONE, 2011
- Markers in the 15q24 nicotinic receptor subunit gene cluster (CHRNA5‐A3‐B4) predict severity of nicotine addiction and response to smoking cessation therapyNeuropsychiatric Genetics, Part B of the American Journal of Medical Genetics (AJMG), 2011
- Acetylcholine Receptor (AChR) α5 Subunit Variant Associated with Risk for Nicotine Dependence and Lung Cancer Reduces (α4β2)2α5 AChR FunctionMolecular Pharmacology, 2010
- Multiple Independent Loci at Chromosome 15q25.1 Affect Smoking Quantity: a Meta-Analysis and Comparison with Lung Cancer and COPDPLoS Genetics, 2010
- Genome-wide meta-analyses identify multiple loci associated with smoking behaviorNature Genetics, 2010
- Genome-wide association scan of tag SNPs identifies a susceptibility locus for lung cancer at 15q25.1Nature Genetics, 2008
- Biochemical verification of tobacco use and cessationNicotine & Tobacco Research, 2002