Malnutrition Impairs Interferon Signaling Through mTOR and FoxO Pathways in Patients With Chronic Hepatitis C
- 31 July 2011
- journal article
- research article
- Published by Elsevier BV in Gastroenterology
- Vol. 141 (1), 128-140.e2
- https://doi.org/10.1053/j.gastro.2011.03.051
Abstract
No abstract availableThis publication has 20 references indexed in Scilit:
- Protease Inhibitor–Resistant Hepatitis C Virus Mutants With Reduced Fitness From Impaired Production of Infectious VirusGastroenterology, 2011
- La Protein Required for Internal Ribosome Entry Site–Directed Translation Is a Potential Therapeutic Target for Hepatitis C Virus ReplicationThe Journal of Infectious Diseases, 2010
- mTORC1-Mediated Cell Proliferation, But Not Cell Growth, Controlled by the 4E-BPsScience, 2010
- Hepatitis C virus genomic RNA dimerization is mediated via a kissing complex intermediateRNA, 2010
- Automated precolumn derivatization system for analyzing physiological amino acids by liquid chromatography/mass spectrometryBiomedical Chromatography, 2009
- Nutritional Control of Protein Biosynthetic Capacity by Insulin via Myc in DrosophilaCell Metabolism, 2008
- Oral administration of branched-chain amino acids activates the mTOR signal in cirrhotic rat liverHepatology Research, 2005
- Phosphorylation of Serine 256 Suppresses Transactivation by FKHR (FOXO1) by Multiple MechanismsJournal of Biological Chemistry, 2002
- mTOR Interacts with Raptor to Form a Nutrient-Sensitive Complex that Signals to the Cell Growth MachineryCell, 2002
- Two Yeast Homologs of ECA39, a Target for c-Myc Regulation, Code for Cytosolic and Mitochondrial Branched-chain Amino Acid AminotransferasesPublished by Elsevier BV ,1996