Selective vulnerability of neurons to acute toxicity after proteasome inhibitor treatment: Implications for oxidative stress and insolubility of newly synthesized proteins
Open Access
- 1 November 2010
- journal article
- Published by Elsevier BV in Free Radical Biology & Medicine
- Vol. 49 (8), 1290-1297
- https://doi.org/10.1016/j.freeradbiomed.2010.07.014
Abstract
No abstract availableKeywords
This publication has 74 references indexed in Scilit:
- Increased protein hydrophobicity in response to aging and Alzheimer diseaseFree Radical Biology & Medicine, 2010
- Molecular mechanisms of proteasome plasticity in agingMechanisms of Ageing and Development, 2010
- Aging and dietary restriction alter proteasome biogenesis and composition in the brain and liverMechanisms of Ageing and Development, 2009
- Proteasome inhibition induces apoptosis in primary human natural killer cells and suppresses NKp46-mediated cytotoxicityHaematologica-The Hematology Journal, 2009
- Heat shock and oxygen radicals stimulate ubiquitin-dependent degradation mainly of newly synthesized proteinsThe Journal of cell biology, 2008
- The neuronal ubiquitin-proteasome system: Murine models and their neurological phenotypeProgress in Neurobiology, 2008
- Role of proteasomes in diseaseBMC Biochemistry, 2007
- Potential Roles for Ubiquitin and the Proteasome during Ribosome BiogenesisMolecular and Cellular Biology, 2006
- Proteasome inhibition by lactacystin in primary neuronal cells induces both potentially neuroprotective and pro‐apoptotic transcriptional responses: a microarray analysisJournal of Neurochemistry, 2005
- Proteasome inhibition protects HT22 neuronal cells from oxidative glutamate toxicityJournal of Neurochemistry, 2005