Mitochondrial Fusion Protects against Neurodegeneration in the Cerebellum
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- 1 August 2007
- journal article
- Published by Elsevier BV in Cell
- Vol. 130 (3), 548-562
- https://doi.org/10.1016/j.cell.2007.06.026
Abstract
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This publication has 43 references indexed in Scilit:
- Complementation between mouse Mfn1 and Mfn2 protects mitochondrial fusion defects caused by CMT2A disease mutationsThe Journal of cell biology, 2007
- OPA1 Controls Apoptotic Cristae Remodeling Independently from Mitochondrial FusionCell, 2006
- Release of OPA1 during Apoptosis Participates in the Rapid and Complete Release of Cytochrome c and Subsequent Mitochondrial FragmentationJournal of Biological Chemistry, 2005
- The GTPase dMiro Is Required for Axonal Transport of Mitochondria to Drosophila SynapsesNeuron, 2005
- Disruption of Fusion Results in Mitochondrial Heterogeneity and DysfunctionJournal of Biological Chemistry, 2005
- Cerebellar granule cell Cre recombinase expressiongenesis, 2003
- Loss of OPA1 Perturbates the Mitochondrial Inner Membrane Structure and Integrity, Leading to Cytochrome c Release and ApoptosisJournal of Biological Chemistry, 2003
- Nuclear gene OPA1, encoding a mitochondrial dynamin-related protein, is mutated in dominant optic atrophyNature Genetics, 2000
- OPA1, encoding a dynamin-related GTPase, is mutated in autosomal dominant optic atrophy linked to chromosome 3q28Nature Genetics, 2000
- The candelabrum cell: A new interneuron in the cerebellar cortexJournal of Comparative Neurology, 1994