Polyglutamine-Expanded Human Huntingtin Transgenes Induce Degeneration of Drosophila Photoreceptor Neurons
Open Access
- 1 September 1998
- journal article
- research article
- Published by Elsevier BV in Neuron
- Vol. 21 (3), 633-642
- https://doi.org/10.1016/s0896-6273(00)80573-5
Abstract
No abstract availableKeywords
This publication has 72 references indexed in Scilit:
- Truncated N-terminal fragments of huntingtin with expanded glutamine repeats form nuclear and cytoplasmic aggregates in cell cultureHuman Molecular Genetics, 1998
- Blocking apoptosis prevents blindness in Drosophila retinal degeneration mutantsNature, 1998
- Cloning of the SCA7 gene reveals a highly unstable CAG repeat expansionNature Genetics, 1997
- Formation of Neuronal Intranuclear Inclusions Underlies the Neurological Dysfunction in Mice Transgenic for the HD MutationCell, 1997
- grim, a novel cell death gene in Drosophila.Genes & Development, 1996
- SCA1 transgenic mice: A model for neurodegeneration caused by an expanded CAG trinucleotide repeatCell, 1995
- Control of programmed cell death by the baculovirus genes p35 and iap.Molecular and Cellular Biology, 1994
- Structure and expression of the Huntington's disease gene: Evidence against simple inactivation due to an expanded CAG repeatSomatic Cell and Molecular Genetics, 1994
- Evidence for a mechanism predisposing to intergenerational CAG repeat instability in spinocerebellar ataxia type INature Genetics, 1993
- Virus-Like Particles in Normal and Tumorous Tissues of DrosophilaScience, 1967