Reduction of Soluble Aβ and Tau, but Not Soluble Aβ Alone, Ameliorates Cognitive Decline in Transgenic Mice with Plaques and Tangles
Open Access
- 1 December 2006
- journal article
- Published by Elsevier BV in Online Journal of Public Health Informatics
- Vol. 281 (51), 39413-39423
- https://doi.org/10.1074/jbc.m608485200
Abstract
No abstract availableThis publication has 44 references indexed in Scilit:
- A specific amyloid-β protein assembly in the brain impairs memoryNature, 2006
- Tau Suppression in a Neurodegenerative Mouse Model Improves Memory FunctionScience, 2005
- Intraneuronal Aβ Causes the Onset of Early Alzheimer’s Disease-Related Cognitive Deficits in Transgenic MiceNeuron, 2005
- Natural oligomers of the amyloid-β protein specifically disrupt cognitive functionNature Neuroscience, 2004
- Dimeric Amyloid β Protein Rapidly Accumulates in Lipid Rafts followed by Apolipoprotein E and Phosphorylated Tau Accumulation in the Tg2576 Mouse Model of Alzheimer's DiseaseJournal of Neuroscience, 2004
- Amygdala Modulation of Memory Consolidation: Interaction with Other Brain SystemsNeurobiology of Learning and Memory, 2002
- Immunization reverses memory deficits without reducing brain Aβ burden in Alzheimer's disease modelNature Neuroscience, 2002
- Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivoNature, 2002
- Soluble Amyloid β Peptide Concentration as a Predictor of Synaptic Change in Alzheimer's DiseaseThe American Journal of Pathology, 1999
- On the structure of microtubules, tau, and paired helical filamentsNeurobiology of Aging, 1995