Casein Kinase 1-dependent Phosphorylation of Familial Advanced Sleep Phase Syndrome-associated Residues Controls PERIOD 2 Stability
Open Access
- 1 April 2011
- journal article
- Published by Elsevier BV
- Vol. 286 (14), 12766-12774
- https://doi.org/10.1074/jbc.m111.224014
Abstract
No abstract availableThis publication has 37 references indexed in Scilit:
- A Conserved Docking Motif for CK1 Binding Controls the Nuclear Localization of NFAT1Molecular and Cellular Biology, 2004
- Posttranslational Regulation of Drosophila PERIOD Protein by Protein Phosphatase 2ACell, 2004
- Drosophila doubletime Mutations Which either Shorten or Lengthen the Period of Circadian Rhythms Decrease the Protein Kinase Activity of Casein Kinase IMolecular and Cellular Biology, 2004
- D4476, a cell‐permeant inhibitor of CK1, suppresses the site‐specific phosphorylation and nuclear exclusion of FOXO1aEMBO Reports, 2004
- The Circadian Gene Period2 Plays an Important Role in Tumor Suppression and DNA Damage Response In VivoCell, 2002
- PNUTS (phosphatase nuclear targeting subunit) inhibits retinoblastoma-directed PP1 activityBiochemical and Biophysical Research Communications, 2002
- An h Per2 Phosphorylation Site Mutation in Familial Advanced Sleep Phase SyndromeScience, 2001
- CASEIN KINASE I: ANOTHER COG IN THE CIRCADIAN CLOCKWORKSChronobiology International, 2001
- Two NoveldoubletimeMutants Alter Circadian Properties and Eliminate the Delay between RNA and Protein inDrosophilaJournal of Neuroscience, 2000
- Resetting of Circadian Time in Peripheral Tissues by Glucocorticoid SignalingScience, 2000