Prenatal cannabinoid exposure and gene expression for neural adhesion molecule L1 in the fetal rat brain
- 1 December 2003
- journal article
- Published by Elsevier BV in Developmental Brain Research
- Vol. 147 (1-2), 201-207
- https://doi.org/10.1016/j.devbrainres.2003.10.016
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- Endocannabinoids in the central nervous system-an overviewProstaglandins, Leukotrienes & Essential Fatty Acids, 2002
- Pharmacology and effects of cannabis: A brief reviewThe British Journal of Psychiatry, 2001
- Regulation of the L1 Cell Adhesion Molecule by Thyroid Hormone in the Developing BrainMolecular and Cellular Neuroscience, 2000
- The endogenous cannabinoid system and brain developmentTrends in Neurosciences, 2000
- Role of endocannabinoids in brain developmentLife Sciences, 1999
- Perinatal Δ9-tetrahydrocannabinol exposure reduces proenkephalin gene expression in the caudate-putamen of adult female ratsLife Sciences, 1998
- Disruption of the mouse L1 gene leads to malformations of the nervous systemNature Genetics, 1997
- The role of cell adhesion molecule L1 in axonal extension, growth cone motility, and signal transduction.Cell and tissue research, 1997
- Changes in tyrosine hydroxylase gene expression in mesencephalic catecholaminergic neurons of immature and adult male rats perinatally exposed to cannabinoidsDevelopmental Brain Research, 1994
- The Consequences of Prenatal Substance Use for the Developing Fetus, Newborn, and Young ChildInternational Journal of the Addictions, 1993