Increased monoaminergic neurotransmission improves compliance with physical activity recommendations in depressed patients with fatigue
- 31 January 2013
- journal article
- Published by Elsevier BV in Medical Hypotheses
- Vol. 80 (1), 47-49
- https://doi.org/10.1016/j.mehy.2012.10.007
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Functional interactions between dopamine, serotonin and norepinephrine neurons: an in-vivo electrophysiological study in rats with monoaminergic lesionsInternational Journal of Neuropsychopharmacology, 2008
- Neuroplasticity of Dopamine Circuits After Exercise: Implications for Central FatigueNeuroMolecular Medicine, 2008
- Noradrenergic Augmentation of Escitalopram Response by Risperidone: Electrophysiologic Studies in the Rat BrainBiological Psychiatry, 2007
- Resolution of Sleepiness and Fatigue in Major Depressive Disorder: A Comparison of Bupropion and the Selective Serotonin Reuptake InhibitorsBiological Psychiatry, 2006
- Empirical Testing of Two Models for Staging Antidepressant Treatment ResistanceJournal of Clinical Psychopharmacology, 2005
- Effect of milnacipran on extracellular monoamine concentrations in the medial prefrontal cortex of rats pre-treated with lithiumEuropean Journal of Pharmacology, 2005
- Residual Symptoms in Depressed Patients Who Respond Acutely to FluoxetinePublished by Physicians Postgraduate Press, Inc ,1999
- Selective serotonin reuptake inhibitors reduce the spontaneous activity of dopaminergic neurons in the ventral tegmental areaBrain Research Bulletin, 1998
- Modulation of the firing activity of noradrenergic neurones in the rat locus coeruleus by the 5‐hydroxtryptamine systemBritish Journal of Pharmacology, 1997
- Biochemical profile of midalcipran (F 2207), 1-phenyl-1-diethyl-aminocarbonyl-2-aminomethyl-cyclopropane (Z) hydrochloride, a potential fourth generation antidepressant drugNeuropharmacology, 1985