Mice with a Partial Deficiency of Manganese Superoxide Dismutase Show Increased Vulnerability to the Mitochondrial Toxins Malonate, 3-Nitropropionic Acid, and MPTP
- 31 January 2001
- journal article
- Published by Elsevier BV in Experimental Neurology
- Vol. 167 (1), 189-195
- https://doi.org/10.1006/exnr.2000.7525
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Partial deficiency of manganese superoxide dismutase exacerbates a transgenic mouse model of amyotrophic lateral sclerosisAnnals of Neurology, 2000
- Nitration of manganese superoxide dismutase in cerebrospinal fluids is a marker for peroxynitrite-mediated oxidative stress in neurodegenerative diseasesAnnals of Neurology, 2000
- Oxidative Damage in Neurodegenerative DiseasesThe Neuroscientist, 1997
- Aging, energy, and oxidative stress in neurodegenerative diseasesAnnals of Neurology, 1995
- 3‐Nitropropionic Acid Neurotoxicity Is Attenuated in Copper/Zinc Superoxide Dismutase Transgenic MiceJournal of Neurochemistry, 1995
- Neurochemical and histologic characterization of striatal excitotoxic lesions produced by the mitochondrial toxin 3-nitropropionic acidJournal of Neuroscience, 1993
- Age‐Dependent Striatal Excitotoxic Lesions Produced by the Endogenous Mitochondrial Inhibitor MalonateJournal of Neurochemistry, 1993
- Do defecs in mitochondrial energy metabolism underlie the pathology of neurodegenerative diseases?Trends in Neurosciences, 1993
- Superoxide DismutasesProgress in Nucleic Acid Research and Molecular Biology, 1991
- Homocysteic acid lesions in rat striatum spare somatostatin—;Neuropeptide Y (NADPH-diaphorase) neuronsNeuroscience Letters, 1990