Cigarette Smoke Silences Innate Lymphoid Cell Function and Facilitates an Exacerbated Type I Interleukin-33-Dependent Response to Infection
- 1 March 2015
- journal article
- research article
- Published by Elsevier BV in Immunity
- Vol. 42 (3), 566-579
- https://doi.org/10.1016/j.immuni.2015.02.011
Abstract
No abstract availableThis publication has 56 references indexed in Scilit:
- Innate lymphoid cells responding to IL-33 mediate airway hyperreactivity independently of adaptive immunityJournal of Allergy and Clinical Immunology, 2011
- IL‐33 synergizes with TCR and IL‐12 signaling to promote the effector function of CD8+ T cellsEuropean Journal of Immunology, 2011
- Innate lymphoid cells mediate influenza-induced airway hyper-reactivity independently of adaptive immunityNature Immunology, 2011
- Experimental Rhinovirus Infection as a Human Model of Chronic Obstructive Pulmonary Disease ExacerbationAmerican Journal of Respiratory and Critical Care Medicine, 2011
- Systemically dispersed innate IL-13–expressing cells in type 2 immunityProceedings of the National Academy of Sciences of the United States of America, 2010
- Nuocytes represent a new innate effector leukocyte that mediates type-2 immunityNature, 2010
- The IL-1-like cytokine IL-33 is inactivated after maturation by caspase-1Proceedings of the National Academy of Sciences of the United States of America, 2009
- IL-33–activated dendritic cells induce an atypical TH2-type responseJournal of Allergy and Clinical Immunology, 2009
- IL-33 exacerbates antigen-induced arthritis by activating mast cellsProceedings of the National Academy of Sciences of the United States of America, 2008
- IL-33 mediates antigen-induced cutaneous and articular hypernociception in miceProceedings of the National Academy of Sciences of the United States of America, 2008