Antiapoptotic Protein Partners Fortilin and MCL1 Independently Protect Cells from 5-Fluorouracil-induced Cytotoxicity
Open Access
- 1 September 2004
- journal article
- Published by Elsevier BV in Online Journal of Public Health Informatics
- Vol. 279 (39), 40868-40875
- https://doi.org/10.1074/jbc.m401454200
Abstract
No abstract availableThis publication has 30 references indexed in Scilit:
- Retinoic Acid Repression of Bone Morphogenetic Protein 4 in Inner Ear DevelopmentMolecular and Cellular Biology, 2003
- Physical and Functional Interaction between Myeloid Cell Leukemia 1 Protein (MCL1) and FortilinOnline Journal of Public Health Informatics, 2002
- Characterization of Fortilin, a Novel Antiapoptotic ProteinOnline Journal of Public Health Informatics, 2001
- Induction of Apoptosis by a Secreted Lipocalin That is Transcriptionally Regulated by IL-3 DeprivationScience, 2001
- Duplexes of 21-nucleotide RNAs mediate RNA interference in cultured mammalian cellsNature, 2001
- RNA interference is mediated by 21- and 22-nucleotide RNAsGenes & Development, 2001
- Regulation of Apoptosis and Cell Cycle Progression by MCL1Journal of Biological Chemistry, 2000
- A common E2F-1 and p73 pathway mediates cell death induced by TCR activationNature, 2000
- cDNA sequence coding for a translationally controlled human tumor proteinNucleic Acids Research, 1989
- Nucleotide sequence of a major messenger RNA for a 21 kilodalton polypeptide that is under translational control in mouse tumor cellsNucleic Acids Research, 1988