A genetic and physiological study of impaired glucose homeostasis control in C57BL/6J mice
- 24 February 2005
- journal article
- research article
- Published by Springer Science and Business Media LLC in Diabetologia
- Vol. 48 (4), 675-686
- https://doi.org/10.1007/s00125-005-1680-z
Abstract
No abstract availableKeywords
This publication has 54 references indexed in Scilit:
- A quantitative trait locus that accounts for glucose intolerance maps to chromosome 8 in hereditary obese KK-Ay miceInternational Journal of Obesity, 2002
- Genetic Analysis of a New Mouse Model for Non-Insulin-Dependent DiabetesGenomics, 2001
- Gene Expression Profile of Aging and Its Retardation by Caloric RestrictionScience, 1999
- Variable Expression of Hepatic Glucokinase in Mice Is Due to a Regulational Locus That Cosegregates with the Glucokinase GeneGenomics, 1997
- Truncation of Kir6.2 produces ATP-sensitive K+ channels in the absence of the sulphonylurea receptorNature, 1997
- The cDNA sequence of proton-pumping nicotinamide nucleotide transhydrogenase from man and mouseBiochimica et Biophysica Acta (BBA) - Bioenergetics, 1996
- Chromosomal mapping of genetic loci associated with non-insulin dependent diabetes in the GK ratNature Genetics, 1996
- Genetic dissection of complex traits: guidelines for interpreting and reporting linkage resultsNature Genetics, 1995
- Proton‐translocating transhydrogenase and NAD‐ and NADP‐linked isocitrate dehydrogenases operate in a substrate cycle which contributes to fine regulation of the tricarboxylic acid cycle activity in mitochondriaFEBS Letters, 1994
- Insulin secretory abnormalities in subjects with hyperglycemia due to glucokinase mutations.JCI Insight, 1994