The Protective Role of Per2 Against Carbon Tetrachloride-Induced Hepatotoxicity
- 31 January 2009
- journal article
- Published by Elsevier BV in The American Journal of Pathology
- Vol. 174 (1), 63-70
- https://doi.org/10.2353/ajpath.2009.080430
Abstract
No abstract availableKeywords
Funding Information
- National Natural Science Foundation of China (30730030)
- Ministry of Education of the People's Republic of China (706031)
This publication has 40 references indexed in Scilit:
- Uncoupling protein-2 induction in rat hepatocytes after acute carbon tetrachloride liver injuryJournal of Cellular Physiology, 2008
- 1α,25‐dihydroxyvitamin D3 attenuates cyanide‐induced neurotoxicity by inhibiting uncoupling protein‐2 up‐regulationJournal of Neuroscience Research, 2008
- A temporal study on the histopathological, biochemical and molecular responses of CCl4-induced hepatotoxicity in Cyp2e1-null miceToxicology, 2006
- PPARα-Mediated Upregulation of Uncoupling Protein-2 Switches Cyanide-Induced Apoptosis to Necrosis in Primary Cortical CellsToxicological Sciences, 2006
- Inactivation of stress protein p8 increases murine carbon tetrachloride hepatotoxicity via preserved CYP2E1 activityHepatology, 2005
- Obesity and Metabolic Syndrome in Circadian Clock Mutant MiceScience, 2005
- Enhancement of Cyanide-Induced Mitochondrial Dysfunction and Cortical Cell Necrosis by Uncoupling Protein-2Toxicological Sciences, 2005
- CLOCK is involved in the circadian transactivation of peroxisome-proliferator-activated receptor α (PPARα) in miceBiochemical Journal, 2005
- Hepatotoxicity and Mechanism of Action of Haloalkanes: Carbon Tetrachloride as a Toxicological ModelCritical Reviews in Toxicology, 2003
- Resistance to Carbon Tetrachloride-Induced Hepatotoxicity in Mice Which Lack CYP2E1 ExpressionToxicology and Applied Pharmacology, 1998