Impaired KLHL3-Mediated Ubiquitination of WNK4 Causes Human Hypertension
Top Cited Papers
Open Access
- 28 March 2013
- journal article
- research article
- Published by Elsevier BV in Cell Reports
- Vol. 3 (3), 858-868
- https://doi.org/10.1016/j.celrep.2013.02.024
Abstract
No abstract availableKeywords
This publication has 42 references indexed in Scilit:
- Activation of the renal Na + :Cl − cotransporter by angiotensin II is a WNK4-dependent processProceedings of the National Academy of Sciences of the United States of America, 2012
- Mutations in kelch-like 3 and cullin 3 cause hypertension and electrolyte abnormalitiesNature, 2012
- The calcineurin inhibitor tacrolimus activates the renal sodium chloride cotransporter to cause hypertensionNature Medicine, 2011
- Ubiquitin Ligase Activity of Cul3-KLHL7 Protein Is Attenuated by Autosomal Dominant Retinitis Pigmentosa Causative MutationOnline Journal of Public Health Informatics, 2011
- PDZ-RhoGEF ubiquitination by Cullin3–KLHL20 controls neurotrophin-induced neurite outgrowthThe Journal of cell biology, 2011
- Downregulation of NCC and NKCC2 cotransporters by kidney-specific WNK1 revealed by gene disruption and transgenic mouse modelsHuman Molecular Genetics, 2010
- Kelch-like homologue 9 mutation is associated with an early onset autosomal dominant distal myopathyBrain, 2010
- The Cullin 3 substrate adaptor KLHL20 mediates DAPK ubiquitination to control interferon responsesThe EMBO Journal, 2010
- Angiotensin II signaling increases activity of the renal Na-Cl cotransporter through a WNK4-SPAK-dependent pathwayProceedings of the National Academy of Sciences of the United States of America, 2009
- Constitutive Turnover of Cyclin E by Cul3 Maintains QuiescenceMolecular and Cellular Biology, 2007