Expression of p16INK4a and other cell cycle regulator and senescence associated genes in aging human kidney
- 1 February 2004
- journal article
- Published by Elsevier BV in Kidney International
- Vol. 65 (2), 510-520
- https://doi.org/10.1111/j.1523-1755.2004.00438.x
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Bypass of telomere-dependent replicative senescence (M1) upon overexpression of Cdk4 in normal human epithelial cellsOncogene, 2003
- Historical claims and current interpretations of replicative agingNature Biotechnology, 2002
- Role of p14ARF in Replicative and Induced Senescence of Human FibroblastsMolecular and Cellular Biology, 2001
- Complex Mechanisms Underlying Impaired Activation of Cdk4 and Cdk2 in Replicative Senescence: Roles of p16, p21, and Cyclin D1Experimental Cell Research, 1999
- The Banff 97 working classification of renal allograft pathologyKidney International, 1999
- Extension of Life-Span by Introduction of Telomerase into Normal Human CellsScience, 1998
- Accumulation of p16INK4a in mouse fibroblasts as a function of replicative senescence and not of retinoblastoma gene statusOncogene, 1997
- Oncogenic ras Provokes Premature Cell Senescence Associated with Accumulation of p53 and p16INK4aCell, 1997
- A new regulatory motif in cell-cycle control causing specific inhibition of cyclin D/CDK4Nature, 1993
- Telomeres shorten during ageing of human fibroblastsNature, 1990