Regulation of the Potential Marker for Intestinal Cells, Bmi1, by β-Catenin and the Zinc Finger Protein KLF4
Open Access
- 1 February 2012
- journal article
- Published by Elsevier BV
- Vol. 287 (6), 3760-3768
- https://doi.org/10.1074/jbc.m111.316349
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Altered intestinal epithelial homeostasis in mice with intestine-specific deletion of the Krüppel-like factor 4 geneDevelopmental Biology, 2011
- Bmi1 is expressed in vivo in intestinal stem cellsNature Genetics, 2008
- Induction of Pluripotent Stem Cells from Adult Human Fibroblasts by Defined FactorsCell, 2007
- Identification of stem cells in small intestine and colon by marker gene Lgr5Nature, 2007
- Mel-18, a Polycomb Group Protein, Regulates Cell Proliferation and Senescence via Transcriptional Repression of Bmi-1 and c-Myc OncoproteinsMolecular Biology of the Cell, 2007
- Elevated Bmi-1 expression is associated with dysplastic cell transformation during oral carcinogenesis and is required for cancer cell replication and survivalBritish Journal of Cancer, 2006
- Induction of Pluripotent Stem Cells from Mouse Embryonic and Adult Fibroblast Cultures by Defined FactorsCell, 2006
- Role of Bmi-1 and Ring1A in H2A Ubiquitylation and Hox Gene SilencingMolecular Cell, 2005
- Role of histone H2A ubiquitination in Polycomb silencingNature, 2004
- Identification of the DNA sequence that interacts with the gut-enriched Kruppel-like factorNucleic Acids Research, 1998