Possible Role of Apoptosis in the Pathogenesis of Bleomycin-Induced Scleroderma
Open Access
- 1 January 2004
- journal article
- Published by Elsevier BV in Journal of Investigative Dermatology
- Vol. 122 (1), 44-50
- https://doi.org/10.1046/j.0022-202x.2003.22121.x
Abstract
No abstract availableKeywords
This publication has 32 references indexed in Scilit:
- Role of apoptosis and transforming growth factor β1 in fibroblast selection and activation in systemic sclerosisArthritis & Rheumatism, 2000
- Opposing Effects of Transmembrane and Soluble FAS Ligand Expression on Inflammation and Tumor Cell SurvivalThe Journal of Experimental Medicine, 2000
- Apoptosis in rheumatic diseasesAmerican Journal Of Medicine, 2000
- The Deletion of Transforming Growth Factor-β-Induced Myofibroblasts Depends on Growth Conditions and Actin OrganizationThe American Journal of Pathology, 1999
- Apoptosis in Human Disease: A New Skin for the Old Ceremony?Biochemical and Biophysical Research Communications, 1999
- Inducible Nonlymphoid Expression of Fas Ligand Is Responsible for Superantigen-Induced Peripheral Deletion of T CellsImmunity, 1998
- The role of FasL-induced apoptosis in immune privilegeImmunology Today, 1997
- Bleomycin induces apoptosis in human alveolar macrophagesAmerican Journal of Physiology-Lung Cellular and Molecular Physiology, 1995
- The polypeptide encoded by the cDNA for human cell surface antigen Fas can mediate apoptosisCell, 1991
- Systemic scleroderma: Clinical and pathophysiologic aspectsJournal of the American Academy of Dermatology, 1988