Tumor necrosis factor-α alters calcium handling and increases arrhythmogenesis of pulmonary vein cardiomyocytes
- 1 March 2007
- journal article
- Published by Elsevier BV in Life Sciences
- Vol. 80 (19), 1806-1815
- https://doi.org/10.1016/j.lfs.2007.02.029
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Effects of a Na+/Ca2+ exchanger inhibitor on pulmonary vein electrical activity and ouabain-induced arrhythmogenicityCardiovascular Research, 2006
- Comparison of ion channel distribution and expression in cardiomyocytes of canine pulmonary veins versus left atriumCardiovascular Research, 2005
- T‐Type Calcium Current in Electrical Activity of Cardiomyocytes Isolated from Rabbit Pulmonary VeinJournal of Cardiovascular Electrophysiology, 2004
- N-Acetylcysteine Prevents the Deleterious Effect of Tumor Necrosis Factor-α on Calcium Transients and Contraction in Adult Rat CardiomyocytesCirculation, 2004
- Sarcoplasmic Reticulum Ca 2+ and Heart FailureCirculation Research, 2003
- Pacing-Induced Spontaneous Activity in Myocardial Sleeves of Pulmonary Veins After Treatment With RyanodineCirculation, 2003
- Tumor necrosis factor alpha and interleukin 1beta are responsible for in vitro myocardial cell depression induced by human septic shock serum.The Journal of Experimental Medicine, 1996
- TNFα receptor expression in rat cardiac myocytes: TNFα inhibition of L‐type Ca2+ current and Ca2+ transientsFEBS Letters, 1995
- Cytokines in Acute Myocardial InfarctionJournal of Cardiovascular Pharmacology, 1994
- Cellular basis for the negative inotropic effects of tumor necrosis factor-alpha in the adult mammalian heart.JCI Insight, 1993