Novel mRNA isoforms of the sodium channels Nav1.2, Nav1.3 and Nav1.7 encode predicted two-domain, truncated proteins
- 26 August 2008
- journal article
- Published by Elsevier BV in Neuroscience
- Vol. 155 (3), 797-808
- https://doi.org/10.1016/j.neuroscience.2008.04.060
Abstract
No abstract availableKeywords
This publication has 84 references indexed in Scilit:
- Characterization of 5′ untranslated regions of the voltage-gated sodium channels SCN1A, SCN2A, and SCN3A and identification of cis-conserved noncoding sequencesGenomics, 2007
- Characterising alternate splicing and tissue specific expression in the chicken from ESTsCytogenetic and Genome Research, 2007
- The sodium channel Nav1.5a is the predominant isoform expressed in adult mouse dorsal root ganglia and exhibits distinct inactivation properties from the full-length Nav1.5 channelMolecular and Cellular Neuroscience, 2007
- A childhood epilepsy mutation reveals a role for developmentally regulated splicing of a sodium channelMolecular and Cellular Neuroscience, 2007
- Three novel ABCC5 splice variants in human retina and their role as regulators of ABCC5 gene expressionBMC Molecular Biology, 2007
- Nova2 Interacts with a Cis-Acting Polymorphism to Influence the Proportions of Drug-Responsive Splice Variants of SCN1AAmerican Journal of Human Genetics, 2007
- Dominant-negative suppression of Cav2.1 currents by α12.1 truncations requires the conserved interaction domain for β subunitsMolecular and Cellular Neuroscience, 2007
- Sequence and comparative analysis of the chicken genome provide unique perspectives on vertebrate evolutionNature, 2004
- Novel Isoforms of the Sodium Channels Nav1.8 and Nav1.5 Are Produced by a Conserved Mechanism in Mouse and RatOnline Journal of Public Health Informatics, 2004
- Evidence for the widespread coupling of alternative splicing and nonsense-mediated mRNA decay in humansProceedings of the National Academy of Sciences of the United States of America, 2002