PGC-1α Activates CYP7A1 and Bile Acid Biosynthesis
Open Access
- 1 December 2003
- journal article
- Published by Elsevier BV
- Vol. 278 (50), 50047-50052
- https://doi.org/10.1074/jbc.m309736200
Abstract
No abstract availableThis publication has 19 references indexed in Scilit:
- Coordinated Control of Cholesterol Catabolism to Bile Acids and of Gluconeogenesis via a Novel Mechanism of Transcription Regulation Linked to the Fasted-to-fed CycleOnline Journal of Public Health Informatics, 2003
- The Enzymes, Regulation, and Genetics of Bile Acid SynthesisAnnual Review of Biochemistry, 2003
- Transport of Cholesterol into Mitochondria Is Rate-limiting for Bile Acid Synthesis via the Alternative Pathway in Primary Rat HepatocytesOnline Journal of Public Health Informatics, 2002
- Cytokine Stimulation of Energy Expenditure through p38 MAP Kinase Activation of PPARγ Coactivator-1Molecular Cell, 2001
- The Peroxisome Proliferator-activated Receptor α (PPARα) Regulates Bile Acid BiosynthesisPublished by Elsevier BV ,2000
- One or More Labile Proteins Regulate the Stability of Chimeric mRNAs Containing the 3′-Untranslated Region of Cholesterol-7α-hydroxylase mRNAPublished by Elsevier BV ,2000
- The Coactivator PGC-1 Cooperates with Peroxisome Proliferator-Activated Receptor α in Transcriptional Control of Nuclear Genes Encoding Mitochondrial Fatty Acid Oxidation EnzymesMolecular and Cellular Biology, 2000
- Gallstones and diabetes: A case-control study in a free-living population sampleHepatology, 1997
- Insulin suppresses bile acid synthesis in cultured rat hepatocytes by down-regulation of cholesterol 27-hydroxylase and sterol 7?-hydroxylase gene transcription*1, *2Hepatology, 1995
- ALTERED BILE ACID METABOLISM IN ALLOXAN DIABETIC RATSThe Japanese Journal of Pharmacology, 1979