High TGFβ-Smad Activity Confers Poor Prognosis in Glioma Patients and Promotes Cell Proliferation Depending on the Methylation of the PDGF-B Gene
Open Access
- 13 February 2007
- journal article
- Published by Elsevier BV in Cancer Cell
- Vol. 11 (2), 147-160
- https://doi.org/10.1016/j.ccr.2006.11.023
Abstract
No abstract availableThis publication has 34 references indexed in Scilit:
- Inhibition of TGFβ signaling in cancer therapyCurrent Opinion in Genetics & Development, 2006
- TLR agonists regulate PDGF-B production and cell proliferation through TGF-β/type I IFN crosstalkThe EMBO Journal, 2005
- The PDGF family: four gene products form five dimeric isoformsCytokine & Growth Factor Reviews, 2004
- Imatinib as a Paradigm of Targeted TherapiesAdvances in Cancer Research, 2004
- Smad-dependent and Smad-independent pathways in TGF-β family signallingNature, 2003
- Targeting the TGFβ signaling network in human neoplasiaCancer Cell, 2003
- Platelet-Derived Growth Factor-B Enhances Glioma Angiogenesis by Stimulating Vascular Endothelial Growth Factor Expression in Tumor Endothelia and by Promoting Pericyte RecruitmentThe American Journal of Pathology, 2003
- TGF-β signaling in tumor suppression and cancer progressionNature Genetics, 2001
- PDGF autocrine stimulation dedifferentiates cultured astrocytes and induces oligodendrogliomas and oligoastrocytomas from neural progenitors and astrocytes in vivoGenes & Development, 2001
- Gliomagenesis: genetic alterations and mouse modelsNature Reviews Genetics, 2001