Cyanide-Induced Apoptosis Involves Oxidative-Stress-Activated NF-κB in Cortical Neurons
- 15 April 2000
- journal article
- Published by Elsevier BV in Toxicology and Applied Pharmacology
- Vol. 164 (2), 196-205
- https://doi.org/10.1006/taap.2000.8900
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- Cyanide metabolism in motor neuron diseaseActa Neurologica Scandinavica, 2009
- Differential Susceptibility of Brain Areas to Cyanide Involves Different Modes of Cell DeathToxicology and Applied Pharmacology, 1999
- Ca2+‐Mediated Activation of c‐Jun N‐Terminal Kinase and Nuclear Factor κB by NMDA in Cortical Cell CulturesJournal of Neurochemistry, 1998
- Caspase-3 Is Required for DNA Fragmentation and Morphological Changes Associated with ApoptosisJournal of Biological Chemistry, 1998
- Sequential activation of ICE-like and CPP32-like proteases during Fas-mediated apoptosisNature, 1996
- Cyanide-stimulated inositol 1,4,5-trisphosphate formation: An intracellular neurotoxic signaling cascadeJournal of Biochemical Toxicology, 1996
- Sequential reduction of mitochondrial transmembrane potential and generation of reactive oxygen species in early programmed cell death.The Journal of Experimental Medicine, 1995
- Blockade of N-methyl-d-aspartate receptors prevents cyanide-induced neuronal injury in primary hippocampal culturesToxicology and Applied Pharmacology, 1992
- Multiple nuclear factors interact with the immunoglobulin enhancer sequencesCell, 1986
- Rat cortical neurons in cell culture: Culture methods, cell morphology, electrophysiology, and synapse formationBrain Research, 1978