The vitamin K cycle
- 1 August 2005
- journal article
- review article
- Published by Elsevier BV in Journal of Thrombosis and Haemostasis
- Vol. 3 (8), 1873-1878
- https://doi.org/10.1111/j.1538-7836.2005.01419.x
Abstract
No abstract availableThis publication has 34 references indexed in Scilit:
- Chemical Modification of Cysteine Residues Is a Misleading Indicator of Their Status as Active Site Residues in the Vitamin K-dependent γ-Glutamyl Carboxylation ReactionOnline Journal of Public Health Informatics, 2004
- Compound heterozygous mutations in the γ‐glutamyl carboxylase gene cause combined deficiency of all vitamin K‐dependent blood coagulation factorsBritish Journal of Haematology, 2004
- Identification of the gene for vitamin K epoxide reductaseNature, 2004
- Determination of Disulfide Bond Assignment of Human Vitamin K-dependent γ-Glutamyl Carboxylase by Matrix-assisted Laser Desorption/Ionization Time-of-Flight Mass SpectrometryOnline Journal of Public Health Informatics, 2003
- Validation of Methods for CYP2C9 Genotyping: Frequencies of Mutant Alleles in a Swedish PopulationBiochemical and Biophysical Research Communications, 1999
- Spontaneous calcification of arteries and cartilage in mice lacking matrix GLA proteinNature, 1997
- Processive Post-translational ModificationOnline Journal of Public Health Informatics, 1995
- Vitamin K2 modulates proliferation and function of osteoblastic cells in vitroBiochemical and Biophysical Research Communications, 1992
- Cloning and Expression of the cDNA for Human γ-glutamyl CarboxylaseScience, 1991
- Reduced thioredoxin: A possible physiological cofactor for vitamin k epoxide reductase. further support for an active site disulfideBiochemical and Biophysical Research Communications, 1988