RIG-I and TLR3 are both required for maximum interferon induction by influenza virus in human lung alveolar epithelial cells
- 1 August 2015
- journal article
- Published by Elsevier BV in Virology
- Vol. 482, 181-188
- https://doi.org/10.1016/j.virol.2015.03.048
Abstract
No abstract availableKeywords
Funding Information
- Flight Attendant Medical Research Institute
- National Institutes of Health (1 I01 BX001937)
- National Institute of Allergy and Infectious Diseases (5U19AI062629)
- U.S. Department of Veterans Affairs
This publication has 39 references indexed in Scilit:
- Bacillus anthracis Lethal Toxin Reduces Human Alveolar Epithelial Barrier FunctionInfection and Immunity, 2012
- Lambda Interferon Is the Predominant Interferon Induced by Influenza A Virus Infection In VivoJournal of Virology, 2010
- Innate immune response to H3N2 and H1N1 influenza virus infection in a human lung organ culture modelVirology, 2010
- Activation of innate immune antiviral responses by Nod2Nature Immunology, 2009
- The Intracellular Sensor NLRP3 Mediates Key Innate and Healing Responses to Influenza A Virus via the Regulation of Caspase-1Immunity, 2009
- Small self-RNA generated by RNase L amplifies antiviral innate immunityNature, 2007
- Type III Interferon (IFN) Induces a Type I IFN-Like Response in a Restricted Subset of Cells through Signaling Pathways Involving both the Jak-STAT Pathway and the Mitogen-Activated Protein KinasesJournal of Virology, 2007
- Inhibition of Retinoic Acid-Inducible Gene I-Mediated Induction of Beta Interferon by the NS1 Protein of Influenza A VirusJournal of Virology, 2007
- RIG-I-Mediated Antiviral Responses to Single-Stranded RNA Bearing 5'-PhosphatesScience, 2006
- Identification and Characterization of MAVS, a Mitochondrial Antiviral Signaling Protein that Activates NF-κB and IRF3Cell, 2005