Uncoupling mitochondrial activity maintains body during hemorrhage-induced O2 deficit in the anesthetized rat
- 1 March 2013
- journal article
- Published by Elsevier BV in Respiratory Physiology & Neurobiology
- Vol. 186 (1), 87-94
- https://doi.org/10.1016/j.resp.2012.12.006
Abstract
No abstract availableKeywords
This publication has 53 references indexed in Scilit:
- Oxygen deficit and H2S in hemorrhagic shock in ratsCritical Care, 2012
- Mitochondrial proton and electron leaksEssays in Biochemistry, 2010
- Hypoxia reduces the hypothalamic thermogenic threshold and thermosensitivityThe Journal of Physiology, 2009
- UCP2, not a physiologically relevant uncoupler but a glucose sparing switch impacting ROS production and glucose sensingBiochimica et Biophysica Acta (BBA) - Bioenergetics, 2009
- Free radicals run in lizard families: a mitochondrial uncoupling phenomenon or not?Biology Letters, 2009
- Trauma-hemorrhagic shock-induced pulmonary epithelial and endothelial cell injury utilizes different programmed cell death signaling pathwaysAmerican Journal of Physiology-Lung Cellular and Molecular Physiology, 2009
- Why are mitochondria involved in apoptosis? Permeability transition pores and apoptosis as selective mechanisms to eliminate superoxide‐producing mitochondria and cellFEBS Letters, 1996
- Oxygen uptake/oxygen supply dependency: Fact or fiction?Acta Anaesthesiologica Scandinavica, 1995
- Metabolism and ventilation in hypoxic rats: effect of body massRespiration Physiology, 1994
- Ventilatory and metabolic responses to cold and CO-induced hypoxia in awake ratsRespiration Physiology, 1994