Deficits in ERK and CREB activation in the hippocampus after traumatic brain injury
- 7 August 2009
- journal article
- research article
- Published by Elsevier BV in Neuroscience Letters
- Vol. 459 (2), 52-56
- https://doi.org/10.1016/j.neulet.2009.04.064
Abstract
No abstract availableKeywords
This publication has 51 references indexed in Scilit:
- Persistent working memory dysfunction following traumatic brain injury: Evidence for a time-dependent mechanismNeuroscience, 2009
- Acute activation of mitogen-activated protein kinases following traumatic brain injury in the rat: implications for posttraumatic cell deathExperimental Neurology, 2003
- There Is Differential Loss of Pyramidal Cells from the Human Hippocampus with Survival after Blunt Head InjuryJournal of Neuropathology and Experimental Neurology, 2003
- The role of extracellular signal-regulated kinase in cognitive and motor deficits following experimental traumatic brain injuryNeuroscience, 2002
- Quantitative structural changes in white and gray matter 1 year following traumatic brain injury in ratsActa Neuropathologica, 2002
- Differential Activation of Mitogen-Activated Protein Kinase Pathways after Traumatic Brain Injury in the Rat HippocampusJournal of Cerebral Blood Flow & Metabolism, 2002
- N‐methyl‐D‐aspartate receptors: Transient loss of NR1/NR2A/NR2B subunits after traumatic brain injury in a rodent modelJournal of Neuroscience Research, 2002
- Temporal Lobe Morphology in Normal Aging and Traumatic Brain Injury2002
- Age-Dependency of45Calcium Accumulation Following Lateral Fluid Percussion: Acute and Delayed PatternsJournal of Neurotrauma, 2001
- Chronic failure in the maintenance of long-term potentiation following fluid percussion injury in the ratBrain Research, 2000