Smad2 Protein Disruption in the Central Nervous System Leads to Aberrant Cerebellar Development and Early Postnatal Ataxia in Mice
Open Access
- 1 May 2011
- journal article
- Published by Elsevier BV in Online Journal of Public Health Informatics
- Vol. 286 (21), 18766-18774
- https://doi.org/10.1074/jbc.m111.223271
Abstract
No abstract availableKeywords
This publication has 50 references indexed in Scilit:
- SMAD proteins control DROSHA-mediated microRNA maturationNature, 2008
- Behavioural characterisation of the robotic mouse mutantBehavioural Brain Research, 2007
- Bioluminescence imaging of Smad signaling in living mice shows correlation with excitotoxic neurodegenerationProceedings of the National Academy of Sciences of the United States of America, 2006
- Altered subcellular location of phosphorylated Smads in Alzheimer's diseaseEuropean Journal of Neuroscience, 2006
- Ectopic expression of phospho‐Smad2 in Alzheimer's disease: Uncoupling of the transforming growth factor‐β pathway?Journal of Neuroscience Research, 2006
- SMAD pathway mediation of BDNF and TGFβ2 regulation of proliferation and differentiation of hippocampal granule neuronsDevelopment, 2005
- Mice exclusively expressing the short isoform of Smad2 develop normally and are viable and fertileGenes & Development, 2005
- Transient expression of GABAA receptor α2 and α3 subunits in differentiating cerebellar neuronsDevelopmental Brain Research, 2004
- Transforming growth factor type β1 modulates the effects of basic fibroblast growth factor on growth and phenotypic expression of rat astroblasts in vitroJournal of Cellular Physiology, 1990
- An autoradiographic study of the time of origin and the pattern of granule cell migration in the dentate gyrus of the ratJournal of Comparative Neurology, 1975