Effects of cromakalim, RP49356, diazoxide, glibenclamide and galanin in rat portal vein
- 6 November 1990
- journal article
- research article
- Published by Elsevier BV in European Journal of Pharmacology
- Vol. 190 (1-2), 75-84
- https://doi.org/10.1016/0014-2999(90)94114-d
Abstract
No abstract availableKeywords
This publication has 16 references indexed in Scilit:
- The effects of cromakalim on ATP‐sensitive potassium channels in insulin‐secreting cellsBritish Journal of Pharmacology, 1990
- Glibenclamide is a competitive antagonist of cromakalim, pinacidil and RP 49356 in guinea-pig pulmonary arteryEuropean Journal of Pharmacology, 1989
- Inhibition by glibenclamide of the vasorelaxant action of cromakalim in the ratBritish Journal of Pharmacology, 1989
- Cromakalim (BRL 34915) and diazoxide activate ATP-regulated potassium channels in insulin-secreting cellsPflügers Archiv - European Journal of Physiology, 1989
- Contribution of galanin to non-cholinergic, non-adrenergic transmission in rat ileumBritish Journal of Pharmacology, 1988
- The potassium channel opener cromakalim (BRL 34915) activates ATP-dependent K+ channels in isolated cardiac myocytesBiochemical and Biophysical Research Communications, 1988
- Comparison of the effects of BRL 34915 and verapamil on electrical and mechanical activity in rat portal veinBritish Journal of Pharmacology, 1986
- ATP-sensitive inward rectifier and voltage- and calcium-activated K+ channels in cultured pancreatic islet cellsThe Journal of Membrane Biology, 1985
- ATP-regulated K+ channels in cardiac muscleNature, 1983
- SOME EFFECTS OF SODIUM NITROPRUSSIDE, METHOXYVERAPAMIL (D600) AND NIFEDIPINE ON RAT PORTAL VEINBritish Journal of Pharmacology, 1980