Macrophage phenotypic switch orchestrates the inflammation and repair/regeneration following acute pancreatitis injury
Open Access
- 31 July 2020
- journal article
- research article
- Published by Elsevier BV in EBioMedicine
- Vol. 58, 102920
- https://doi.org/10.1016/j.ebiom.2020.102920
Abstract
No abstract availableKeywords
Funding Information
- Shanghai Municipal Education Commission
This publication has 47 references indexed in Scilit:
- Interleukin-4- and Interleukin-13-Mediated Alternatively Activated Macrophages: Roles in Homeostasis and DiseaseAnnual Review of Immunology, 2013
- Activation of Natural Killer T Cells Promotes M2 Macrophage Polarization in Adipose Tissue and Improves Systemic Glucose Tolerance via Interleukin-4 (IL-4)/STAT6 Protein Signaling Axis in ObesityOnline Journal of Public Health Informatics, 2012
- EZH2 couples pancreatic regeneration to neoplastic progressionGenes & Development, 2012
- Pulse exposure of haematopoietic grafts to prostaglandin E2in vitro facilitates engraftment and recoveryCell Proliferation, 2011
- Macrophage Dysfunction Impairs Resolution of Inflammation in the Wounds of Diabetic MicePLOS ONE, 2010
- Essential role of monocytes and macrophages in the progression of acute pancreatitisWorld Journal of Gastroenterology, 2010
- Role of macrophages in the progression of acute pancreatitisWorld Journal of Gastrointestinal Pharmacology and Therapeutics, 2010
- Hedgehog Signaling Is Required for Effective Regeneration of Exocrine PancreasGastroenterology, 2008
- Wound repair and regenerationNature, 2008
- Preexisting pancreatic acinar cells contribute to acinar cell, but not islet β cell, regenerationJCI Insight, 2007