Enhanced Hyaluronan Signaling and Autophagy Dysfunction by VPS35 D620N
Open Access
- 10 August 2020
- journal article
- research article
- Published by Elsevier BV in Neuroscience
- Vol. 441, 33-45
- https://doi.org/10.1016/j.neuroscience.2020.06.009
Abstract
No abstract availableFunding Information
- National Institute of Neurological Disorders and Stroke (R15NS096702)
- Boise State University
- National Institutes of Health
- National Institute of General Medical Sciences (P20GM103408, P20GM109095)
This publication has 43 references indexed in Scilit:
- Phenotypic Characterization of Retinoic Acid Differentiated SH-SY5Y Cells by Transcriptional ProfilingPLOS ONE, 2013
- Differential gene and transcript expression analysis of RNA-seq experiments with TopHat and CufflinksNature Protocols, 2012
- A Mutation in VPS35, Encoding a Subunit of the Retromer Complex, Causes Late-Onset Parkinson DiseaseAmerican Journal of Human Genetics, 2011
- VPS35 Mutations in Parkinson DiseaseAmerican Journal of Human Genetics, 2011
- Regulation Mechanisms and Signaling Pathways of AutophagyAnnual Review of Genetics, 2009
- GOLPH3 modulates mTOR signalling and rapamycin sensitivity in cancerNature, 2009
- A novel signaling pathway impact analysisBioinformatics, 2008
- Autophagy in the Pathogenesis of DiseaseCell, 2008
- Gene set enrichment analysis: A knowledge-based approach for interpreting genome-wide expression profilesProceedings of the National Academy of Sciences of the United States of America, 2005
- PGC-1α-responsive genes involved in oxidative phosphorylation are coordinately downregulated in human diabetesNature Genetics, 2003