GR/HDAC2/TGFβR1 pathway contributes to prenatal caffeine induced-osteoarthritis susceptibility in male adult offspring rats
- 19 March 2020
- journal article
- research article
- Published by Elsevier BV in Food and Chemical Toxicology
- Vol. 140, 111279
- https://doi.org/10.1016/j.fct.2020.111279
Abstract
No abstract availableKeywords
Funding Information
- National Natural Science Foundation of China
This publication has 41 references indexed in Scilit:
- Glucocorticoid excess and the developmental origins of disease: Two decades of testing the hypothesis – 2012 Curt Richter Award WinnerPsychoneuroendocrinology, 2013
- Caffeine-Induced Activated Glucocorticoid Metabolism in the Hippocampus Causes Hypothalamic-Pituitary-Adrenal Axis Inhibition in Fetal RatsPLOS ONE, 2012
- Dynamic Interaction of HDAC1 with a Glucocorticoid Receptor-regulated Gene Is Modulated by the Activity State of the PromoterOnline Journal of Public Health Informatics, 2011
- A review of the epidemiologic evidence concerning the reproductive health effects of caffeine consumption: A 2000–2009 updateFood and Chemical Toxicology, 2010
- Hypertrophic differentiation of chondrocytes in osteoarthritis: the developmental aspect of degenerative joint disordersArthritis Research & Therapy, 2010
- Epigenetic controlJournal of Cellular Physiology, 2009
- Assessing caffeine exposure in pregnant womenBritish Journal of Nutrition, 2008
- Dose translation from animal to human studies revisitedThe FASEB Journal, 2007
- Mechanisms underlying the role of glucocorticoids in the early life programming of adult diseaseClinical Science, 2007
- Maternal Genistein Alters Coat Color and Protects
A
vy
Mouse Offspring from Obesity by Modifying the Fetal EpigenomeEnvironmental Health Perspectives, 2006