Blastic transformation of BCR‐ABL1 positive chronic myeloid leukaemia through acquisition of CBFB‐MYH11 and mutant KIT
Open Access
- 24 June 2020
- journal article
- letter
- Published by Wiley in British Journal of Haematology
- Vol. 190 (6), e339-e343
- https://doi.org/10.1111/bjh.16904
Abstract
No abstract availableFunding Information
- Deutsche Krebshilfe (111210)
This publication has 16 references indexed in Scilit:
- High-risk additional chromosomal abnormalities at low blast counts herald death by CMLLeukemia, 2020
- Molecular Genetic Characterization of Individual Cancer Cells Isolated via Single-Cell PrintingPLOS ONE, 2016
- Risk stratification of chromosomal abnormalities in chronic myelogenous leukemia in the era of tyrosine kinase inhibitor therapyBlood, 2016
- Comprehensive mutational profiling of core binding factor acute myeloid leukemiaBlood, 2016
- Single cell genotyping of exome sequencing-identified mutations to characterize the clonal composition and evolution of inv(16) AML in a CBL mutated clonal hematopoiesisLeukemia Research, 2016
- inv(16)/t(16;16) acute myeloid leukemia with non–type A CBFB-MYH11 fusions associate with distinct clinical and genetic features and lack KIT mutationsBlood, 2013
- Secondary genetic lesions in acute myeloid leukemia with inv(16) or t(16;16): a study of the German-Austrian AML Study Group (AMLSG)Blood, 2013
- Impact of additional cytogenetic aberrations at diagnosis on prognosis of CML: long-term observation of 1151 patients from the randomized CML Study IVBlood, 2011
- inv(16)(p13q22) in Chronic Myelogenous Leukemia in Blast PhaseAmerican Journal of Clinical Pathology, 2005
- KIT exon 8 mutations associated with core-binding factor (CBF)–acute myeloid leukemia (AML) cause hyperactivation of the receptor in response to stem cell factorBlood, 2005