Reply to Thomson: Exposure to Active and Passive Tobacco Smoke on Urinary Eicosanoid Metabolites in Type 2 Asthma
Open Access
- 1 May 2021
- journal article
- letter
- Published by American Thoracic Society in American Journal of Respiratory and Critical Care Medicine
- Vol. 203 (9), 1204-1205
- https://doi.org/10.1164/rccm.202101-0208le
Abstract
No abstract availableThis publication has 5 references indexed in Scilit:
- Urinary Leukotriene E4and Prostaglandin D2Metabolites Increase in Adult and Childhood Severe Asthma Characterized by Type 2 Inflammation. A Clinical Observational StudyAmerican Journal of Respiratory and Critical Care Medicine, 2021
- Arachidonic acid metabolites and enzyme transcripts in asthma are altered by cigarette smokingAllergy, 2014
- Clinical outcomes and inflammatory biomarkers in current smokers and exsmokers with severe asthmaJournal of Allergy and Clinical Immunology, 2013
- Urinary leukotriene E4 levels identify children with tobacco smoke exposure at risk for asthma exacerbationJournal of Allergy and Clinical Immunology, 2011
- Levels of Prostaglandin E Metabolite, the Major Urinary Metabolite of Prostaglandin E2, Are Increased in SmokersClinical Cancer Research, 2005