Impact of myeloid RIPK1 gene deletion on atherogenesis in ApoE-deficient mice
- 23 February 2021
- journal article
- research article
- Published by Elsevier BV in Atherosclerosis
- Vol. 322, 51-60
- https://doi.org/10.1016/j.atherosclerosis.2021.02.021
Abstract
No abstract availableKeywords
Funding Information
- FWO
- Foundation Against Cancer
This publication has 53 references indexed in Scilit:
- Loss of Cellular Inhibitor of Apoptosis Protein 2 Reduces Atherosclerosis in Atherogenic apoE −/− C57BL/6 Mice on High‐Fat DietJournal of the American Heart Association, 2013
- Phosphorylation-Driven Assembly of the RIP1-RIP3 Complex Regulates Programmed Necrosis and Virus-Induced InflammationCell, 2009
- TNF-α Induces Two Distinct Caspase-8 Activation PathwaysCell, 2008
- Ubiquitination of RIP1 Regulates an NF-κB-Independent Cell-Death Switch in TNF SignalingCurrent Biology, 2007
- Increased Expression of the DNA-Binding Cytokine HMGB1 in Human Atherosclerotic LesionsArteriosclerosis, Thrombosis, and Vascular Biology, 2004
- Catalytic activity of caspase-3 is required for its degradation: stabilization of the active complex by synthetic inhibitorsCell Death & Differentiation, 2004
- NF-κB Antiapoptosis: Induction of TRAF1 and TRAF2 and c-IAP1 and c-IAP2 to Suppress Caspase-8 ActivationScience, 1998
- The Death Domain Kinase RIP Mediates the TNF-Induced NF-κB SignalImmunity, 1998
- Nitric Oxide Reversibly Inhibits Seven Members of the Caspase Family via S-NitrosylationBiochemical and Biophysical Research Communications, 1997
- Chemical composition and physical state of lipid deposits in atherosclerosisAtherosclerosis, 1985