Interleukin-22 exacerbates angiotensin II-induced hypertensive renal injury
- 11 May 2022
- journal article
- research article
- Published by Elsevier BV in International Immunopharmacology
- Vol. 109, 108840
- https://doi.org/10.1016/j.intimp.2022.108840
Abstract
No abstract availableKeywords
Funding Information
- Natural Science Foundation of Hunan Province (2018JJ3818, 2019JJ20035)
- National Natural Science Foundation of China (81401227, 81500559)
This publication has 34 references indexed in Scilit:
- JAK-STAT and the renin-angiotensin systemJAK-STAT, 2012
- Interleukin‐22 promotes osteoclastogenesis in rheumatoid arthritis through induction of RANKL in human synovial fibroblastsArthritis & Rheumatism, 2011
- Functional Biology of the IL-22-IL-22R Pathway in Regulating Immunity and Inflammation at Barrier SurfacesAdvances in Immunology, 2010
- Identification of a human helper T cell population that has abundant production of interleukin 22 and is distinct from TH-17, TH1 and TH2 cellsNature Immunology, 2009
- Innate and Adaptive Interleukin-22 Protects Mice from Inflammatory Bowel DiseaseImmunity, 2008
- The Intrarenal Renin-Angiotensin System: From Physiology to the Pathobiology of Hypertension and Kidney DiseasePharmacological Reviews, 2007
- Role of the T cell in the genesis of angiotensin II–induced hypertension and vascular dysfunctionThe Journal of Experimental Medicine, 2007
- Interleukin 22 (IL-22) plays a protective role in T cell-mediated murine hepatitis: IL-22 is a survival factor for hepatocytes via STAT3 activationJournal of Hepatology, 2004
- TGF-β1–Mediated Inhibition of HK-2 Cell MigrationJournal of the American Society of Nephrology, 2003
- Interleukin-22 (IL-22) Activates the JAK/STAT, ERK, JNK, and p38 MAP Kinase Pathways in a Rat Hepatoma Cell LineOnline Journal of Public Health Informatics, 2002