Dihydromyricetin alleviates doxorubicin-induced cardiotoxicity by inhibiting NLRP3 inflammasome through activation of SIRT1
- 27 February 2020
- journal article
- research article
- Published by Elsevier BV in Biochemical Pharmacology
- Vol. 175, 113888
- https://doi.org/10.1016/j.bcp.2020.113888
Abstract
No abstract availableKeywords
Funding Information
- National Natural Science Foundation of China (81873120)
- Medical and Health Research Program of Zhejiang (2018KY172)
- Public Welfare Technology and Social Development Project of Zhejiang (LGF18H020009)
- Youth Fund General Project of Shaoxing People’s Hospital (2018YB24)
- Research, Development and Popularization of Core Technology for Major Cardiovascular Diseases (LGD19H110002)
This publication has 49 references indexed in Scilit:
- Attenuating effects of dihydromyricetin on angiotensin II-induced rat cardiomyocyte hypertrophy related to antioxidative activity in a NO-dependent mannerPharmaceutical Biology, 2014
- Critical role of caspase-1 in vascular inflammation and development of atherosclerosis in Western diet-fed apolipoprotein E-deficient miceBiochemical and Biophysical Research Communications, 2012
- Emerging roles of SIRT1 deacetylase in regulating cardiomyocyte survival and hypertrophyJournal of Molecular and Cellular Cardiology, 2011
- Doxorubicin and daunorubicin induce processing and release of interleukin-1β through activation of the NLRP3 inflammasomeCancer Biology & Therapy, 2011
- Recombinant human interleukin-1 receptor antagonist protects mice against acute doxorubicin-induced cardiotoxicityEuropean Journal of Pharmacology, 2010
- Resveratrol prevents doxorubicin cardiotoxicity through mitochondrial stabilization and the Sirt1 pathwayFree Radical Biology & Medicine, 2009
- Early Breast Cancer Therapy and Cardiovascular InjuryJournal of the American College of Cardiology, 2007
- Silent Information Regulator 2α, a Longevity Factor and Class III Histone Deacetylase, Is an Essential Endogenous Apoptosis Inhibitor in Cardiac MyocytesCirculation Research, 2004
- Apoptosis in Adriamycin Cardiomyopathy and Its Modulation by ProbucolAntioxidants and Redox Signaling, 2001
- Movement of Bax from the Cytosol to Mitochondria during ApoptosisThe Journal of cell biology, 1997