miR-1468-3p Promotes Aging-Related Cardiac Fibrosis
Open Access
- 5 June 2020
- journal article
- research article
- Published by Elsevier BV in Molecular Therapy Nucleic Acids
- Vol. 20, 589-605
- https://doi.org/10.1016/j.omtn.2020.04.001
Abstract
No abstract availableFunding Information
- University of Turku
- Abo Akademi University
- Biocenter Finland
- Academy of Finland (131020, 297094, 268505)
- Finnish Foundation for Cardiovascular Research
- Jane and Aatos Erkko Foundation
This publication has 54 references indexed in Scilit:
- TGF-β signaling alters H4K20me3 status via miR-29 and contributes to cellular senescence and cardiac agingNature Communications, 2018
- Interventions to promote cardiometabolic health and slow cardiovascular ageingNature Reviews Cardiology, 2018
- Fibroblast-specific TGF-β–Smad2/3 signaling underlies cardiac fibrosisJCI Insight, 2017
- Reverse Expression of Aging-Associated Molecules through Transfection of miRNAs to Aged MiceMolecular Therapy Nucleic Acids, 2017
- Senescent Cardiac Fibroblast Is Critical for Cardiac Fibrosis after Myocardial InfarctionPLOS ONE, 2013
- Unrestrained p38 MAPK Activation in Dusp1/4 Double-Null Mice Induces CardiomyopathyCirculation Research, 2013
- Evolution of microRNA diversity and regulation in animalsNature Reviews Genetics, 2011
- Distinct regulation of B-type natriuretic peptide transcription by p38 MAPK isoformsMolecular and Cellular Endocrinology, 2011
- Many novel mammalian microRNA candidates identified by extensive cloning and RAKE analysisGenome Research, 2006
- Postinfarction Gene Therapy Against Transforming Growth Factor-β Signal Modulates Infarct Tissue Dynamics and Attenuates Left Ventricular Remodeling and Heart FailureCirculation, 2005