Biochemical and Biophysical Research Communications

Journal Information
ISSN / EISSN : 0006291X / 10902104
Current Publisher: Elsevier BV (10.1016)
Former Publisher: Elsevier BV (10.1006)
Total articles ≅ 88,489
Google Scholar h5-index: 64
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Latest articles in this journal

Subroto Chatterjee, Lucy Zheng, Sijia Ma, Djahida Bedja, Veera Venkata Ratnam Bandaru, Grace Kim, Alexa B. Rangecroft, Domenica Iocco, Sean A. Campbell
Biochemical and Biophysical Research Communications; doi:10.1016/j.bbrc.2020.02.104

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Lang Zhao, Kang Fu, Xiaoxing Li, Rui Zhang, Wenjun Wang, Feng Xu, Xiaoping Ji, Yuguo Chen, Chuanbao Li
Biochemical and Biophysical Research Communications; doi:10.1016/j.bbrc.2020.02.096

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Min Zhuang, Qi Lai, Chunju Yang, Yanhua Ma, Baomin Fan, Zhaoxiang Bian, Chengyuan Lin, Jin Bai, Guangzhi Zeng
Biochemical and Biophysical Research Communications; doi:10.1016/j.bbrc.2020.02.023

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Yuu Miyauchi, Ayumi Kurita, Ryohei Yamashita, Tomoyuki Takamatsu, Shin’Ichi Ikushiro, Peter I. MacKenzie, Yoshitaka Tanaka, Yuji Ishii
Biochemical and Biophysical Research Communications; doi:10.1016/j.bbrc.2020.02.075

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Yasuhiro Isono, Mitsuko Furuya, Tatsu Kuwahara, Daisuke Sano, Kae Suzuki, Ryosuke Jikuya, Taku Mitome, Shinji Otake, Takashi Kawahara, Yusuke Ito, et al.
Biochemical and Biophysical Research Communications, Volume 522, pp 931-938; doi:10.1016/j.bbrc.2019.11.184

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Ryohei Sugahara, Wataru Tsuchiya, Toshimasa Yamazaki, Seiji Tanaka, Takahiro Shiotsuki
Biochemical and Biophysical Research Communications, Volume 522, pp 876-880; doi:10.1016/j.bbrc.2019.11.113

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Peng Zhang, Hailong Wang, Yu Chen, Adil Farooq Lodhi, Chunli Sun, Feiyi Sun, Liben Yan, Yulin Deng, Hong Ma
Biochemical and Biophysical Research Communications, Volume 522, pp 910-916; doi:10.1016/j.bbrc.2019.11.161

Abstract:As a cancer treatment strategy, irradiation therapy is widely used that can cause DNA breakage and increase free radicals, which leads to different types of cell death. Among them, apoptosis and autophagy are the most important and the most studied cell death processes. Although the exploration of the relationship between apoptosis and autophagy has been a major area of focus, still the molecular mechanisms of autophagy on apoptosis remain unclear. Here, we have revealed that apoptosis was enhanced by the death receptor 5 (DR5) pathway, and the effect of autophagy on apoptosis was promoted by DR5 interacting with LC3B as well as Caspase8 in gliomas after irradiation. Interestingly, we observed that the addition of four different autophagy inducers, rapamycin (RAP), CCI779, ABT737 and temozolomide (TMZ), induced the differences of DR5 expression and cell apoptosis after irradiation. Unlike RAP and CCI779, ABT737 and TMZ were able to increase DR5 expression and further induce cell death. Therefore, we have concluded that DR5 plays a novel and indispensable role in promoting cell apoptosis under irradiation and suggest a potential therapeutic approach for glioblastoma treatment.
Ying-Xu Shi, Yan-Ji He, Yong Zhou, Hao-Ke Li, Dan Yang, Ren-Yan Li, Zhong-Liang Deng, Yan-Fei Gao
Biochemical and Biophysical Research Communications, Volume 522, pp 924-930; doi:10.1016/j.bbrc.2019.11.182

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Anna Mizutani, Hiroyuki Seimiya
Biochemical and Biophysical Research Communications, Volume 522, pp 945-951; doi:10.1016/j.bbrc.2019.11.191

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Xin Zhang, Natsumi Koga, Hiroyuki Suzuki, Mitsuyasu Kato
Biochemical and Biophysical Research Communications, Volume 522, pp 897-902; doi:10.1016/j.bbrc.2019.11.145

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