Journal of Virology

Journal Information
ISSN / EISSN : 0022-538X / 1098-5514
Published by: American Society for Microbiology (10.1128)
Total articles ≅ 69,634
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Latest articles in this journal

Sara Dochnal, Husain Y. Merchant, Austin R. Schinlever, Aleksandra Babnis, , ,
Journal of Virology, Volume 96;

Herpes simplex virus-1 (HSV-1) enters a latent infection in neurons and periodically reactivates. Reactivation manifests as a variety of clinical symptoms.
Wiyada Wongwiwat, Benjamin Fournier, Irene Bassano, Amr Bayoumy, Claudio Elgueta Karstegl, Christine Styles, Ray Bridges, Christelle Lenoir, David BoutBoul, Despina Moshous, et al.
Journal of Virology, Volume 96;

EBV contributes to several types of human cancer. Some cancers and nonmalignant lymphoproliferative diseases involving T or NK cells contain EBV.
Hainan Liu, Yu Bai, Xun Zhang, Ting Gao, Yue Liu, Entao Li, Xuefeng Wang, Zheng Cao, Lin Zhu, Qincai Dong, et al.
Journal of Virology, Volume 96;

In this study, by in vitro assay and live SARS-CoV-2 virus infection, we provide solid evidence that the SARS-CoV-2 NP associates with G3BP1 and G3BP2 in vitro and in vivo . NP SARS-CoV-2 could efficiently suppress G3BP-mediated SG formation and potentiate viral infection by overcoming antiviral innate immunity mediated by G3BP1 in A549 cell lines and G3BP1 conditional knockout mice ( g3bp1 -cKO) mice, which provide in-depth evidence showing the mechanism underlying NP-related SARS-CoV-2 pathogenesis through G3BPs.
Xusheng Ma, , Zhikuan Luo, Xiaofeng Nian, , , Rui Song, Jingjing Pei, Yanli Huo, Yuanyuan Li, et al.
Journal of Virology, Volume 96;

FMDV is a pathogen that causes one of the world’s most destructive and highly contagious animal diseases. The FMDV 3A protein plays a critical role in viral replication and host range.
Jonas L. Delva, Cliff Van Waesberghe, Wim Van Den Broeck, Jochen A. Lamote, , Sebastiaan Theuns, Liesbeth Couck,
Journal of Virology, Volume 96;

The pseudorabies virus (PRV) vaccine strain Bartha has been and still is critical in the eradication of PRV in numerous countries. However, little is known about how this vaccine strain interacts with host cells and the host immune system.
Aro Kim, Gyeongmin Lee, Ji-Hyeon Hwang, Jong-Hyeon Park, Min Ja Lee, Byounghan Kim,
Journal of Virology, Volume 96;

Early inhibition of foot-and-mouth disease (FMD) virus (FMDV) replication in pigs is highly desirable as FMDV transmission and shedding rates are higher in pigs than in cattle. However, commercial FMD vaccines require at least 4 to 7 days postvaccination (dpv) for protection, and animals are vulnerable to heterologous viruses before acquiring high antibody levels after the second vaccination.
Mengdong Wang, Yun Liu, Chao Qin, Yue Lang, Aotian Xu, Cuilian Yu, Zhan Zhao, Rui Zhang, Jinbo Yang,
Alphaherpesviruses can establish lifelong infections and cause many diseases in humans and animals. Pseudorabies virus (PRV) is a swine alphaherpesvirus that threatens pig production.
Pola Olczak, Ken Matsui, Margaret Wong, Jade Alvarez, Paul Lambert, Neil D. Christensen, , Bettina Huber, Reinhard Kirnbauer, Joshua W. Wang, et al.
Licensed preventive HPV vaccines are composed of VLPs derived by expression of major capsid protein L1. They confer protection generally restricted to infection by the αHPVs targeted by the up-to-9-valent vaccine, and their associated anogenital cancers and genital warts, but do not target βHPV that are associated with CSCC in EV and immunocompromised patients.
Cong Sun, , Chu Xie, Jiu-Feng Sun, Xin-Yan Fang, Yi-Sha Hu, Xiao-Hui Yu, Zheng Liu, ,
The emergence of SARS-CoV-2 variants led to an urgent demand for a broadly effective vaccine against the threat of variant infection. The spike protein S1-based nanoparticle designed in our study could elicit a comprehensive humoral response toward different SARS-CoV-2 variants of concern and variants of interest and will be helpful to combat COVID-19 globally.
Alexandra I. Wells,
Echoviruses are among the most common worldwide causes of aseptic meningitis, which can cause long-term sequelae or even death. The mechanisms by which echoviruses infect the brain are poorly understood, largely owing to the lack of robust in vivo models that recapitulate this aspect of echovirus pathogenesis.
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